{"id":609,"date":"2026-03-12T16:32:13","date_gmt":"2026-03-12T16:32:13","guid":{"rendered":"https:\/\/blogs.imperial.ac.uk\/doii-staff\/?p=609"},"modified":"2026-03-13T16:04:38","modified_gmt":"2026-03-13T16:04:38","slug":"meet-dr-shuchi-agrawal-singh-assistant-professor-in-haematology","status":"publish","type":"post","link":"https:\/\/blogs.imperial.ac.uk\/doii-staff\/2026\/03\/12\/meet-dr-shuchi-agrawal-singh-assistant-professor-in-haematology\/","title":{"rendered":"Meet Dr Shuchi Agrawal Singh, Assistant Professor in Haematology"},"content":{"rendered":"

\"\"<\/h2>\n

In this Q&A, Dr Shuchi Agrawal Singh discusses her work on epigenetics and biomolecular condensation in acute myeloid leukaemia, and the problem of drug resistance and relapse. She reflects on the rapid technological advances in recent years in her field and shares more about her exciting new work on phase separation.<\/h2>\n

<\/p>\n

\n

Tell us who you are and what do you do?<\/span><\/span><\/h2>\n

I am a non\u2011clinical scientist originally from India, where I completed my undergraduate studies and a master\u2019s in biotechnology. I then moved to Germany to do my PhD in M\u00fcnster, focusing on acute myeloid\u00a0leukemia\u00a0and the molecular mechanisms driving the disease.<\/span>\u00a0<\/span><\/p>\n

From early on, I was\u00a0very interested\u00a0in can<\/span>cer research, which motivated my PhD work. During that time, I became particularly drawn to epigenetics. After completing my PhD, I went to Copenhagen for a postdoc at an institute well\u2011known for epigenetic studies\u00a0Biotech Research & Innovation Centre (BRIC). I trained there\u00a0on epigenetics\u00a0with experts in the field before moving to Cambridge for my second postdoc, returning to blood cancer\u00a0research\u00a0and combining both areas<\/span>\u00a0of epigenetics and acute myeloid\u00a0leukemia.<\/span><\/p>\n

I joined Imperial in 2024, in the Centre for Haematology. I am passionate about understanding<\/span>\u00a0the\u00a0basic epigenetic\u00a0mechanisms driving\u00a0drug-resistance in\u00a0acute myeloid\u00a0leukemia, particularly the epigenetic changes that might be reversible. If genes are misbehaving, epigenetic changes\u00a0<\/span>can sometimes be reverted to correct them, which is what motivates me.<\/span><\/p>\n

Was there a particular moment you knew cancer research was the direction you wanted to follow?<\/span><\/span><\/h2>\n

My interest developed gradually. My master\u2019s project was also in blood cancer research, which drew me in. During my PhD, my work covered two angles. One was related to epigenetic<\/span>s\u00a0DNA methylation\u00a0an<\/span>d the\u00a0<\/span>other to signalling pathways\u00a0by\u00a0Flt3-mutation.\u00a0Epigenetics intrigued me more because of its reversible nature, which offers the potential to correct unwanted changes.<\/span><\/p>\n

That motivation led me to pursue my epigenetics postdoc in Copenhagen, which gave me strong training in the field. I could then apply that knowledge back to cancer biology.<\/span><\/p>\n

What motivates you most in your work?<\/span><\/span><\/h2>\n

One of my goals is to find treatments that can improve the lives of patients with blood cancers\u00a0who are having relapse and became resistant to treatments. I am very motivated by understanding resistance mechanisms and why cancer drugs\u00a0fail to\u00a0cure the disease, and why patients relapse or develop resistance.<\/span>\u00a0<\/span><\/p>\n

Resistance and relapse happen across all cancers, blood cancers and solid tumours. In blood cancers, it is easier to detect and monitor because of blood sampling and bone marrow biopsies, which also means research in the field is more advanced. Epigenetic mechanisms play a role across cancers, so what we find here could help us understand other cancers too.<\/span><\/p>\n

Is there anything else\u00a0<\/span>you\u2019d<\/span>\u00a0like to share about your research interests?<\/span><\/h2>\n

Yes, besides epigenetics, my lab also focuses on phase separation, a very new field that connects biophysics and biology called phase separation.<\/span>\u00a0<\/span>To explain simply: if you put drops of oil into water, the oil separates into droplets, which is phase separation. Something similar happens inside cells, where proteins form condensates to carry out functions.<\/span><\/p>\n

In cancer cells, this process becomes abnormal, and cancer cells exploit these droplets for their benefit. My lab is trying to study how these condensates form in acute leukaemia and how we might dissolve them therapeutically.<\/span>\u00a0<\/span>It\u2019s\u00a0challenging currently because the field is new, techniques are complex, and acceptance is still growing, but\u00a0it’s\u00a0an exciting direction. There are already examples in prostate cancer where certain drugs can dissolve condensates.<\/span>\u00a0<\/span>Imperial\u2019s multidisciplinary environment is ideal for this work, so I am currently collaborating with researchers in chemistry, biophysics, and drug discovery, including the Drug Discovery Hub, which has enabled us to screen 2,000 drugs in one go.<\/span>\u00a0<\/span>My drive is to\u00a0establish\u00a0this emerging area at Imperial and hopefully be among the first to make significant discoveries.<\/span><\/p>\n

Epigenetics has grown so rapidly. How has your own understanding and the field changed since you started?<\/span><\/span><\/h2>\n

The progress in the last 10\u00a0to\u00a015 years has been enormous. Technological advancements have transformed what we can study. When I started my PhD, next\u2011generation sequencing was new and extremely expensive. Now it is widely accessible, with multiplexing reducing the cost.<\/span>\u00a0<\/span><\/p>\n

Single\u2011cell epigenomics has been a revolution and allows us to see heterogeneity in cancer at much higher resolution. We can now detect different populations of cancer cells based on gene expression or epigenetic changes, understand why some survive treatment, and link this to relapse. These tools have given us a much deeper understanding of epigenetics and cancer.<\/span><\/p>\n

You founded a popular Cancer Epigenetics Seminar Series. How did that begin?<\/span><\/span><\/h2>\n

The series during the pandemic. Everyone was at home, and I attended many online webinars, and I thought I should start a seminar series focused on cancer epigenetics to bring two big communities, cancer\u00a0researchers\u00a0and basic epigenetics researchers, together.<\/span>\u00a0<\/span><\/p>\n

Clinical cancer researchers often lack deep exposure to the basic science of epigenetics, while epigenetic scientists may not work on disease models. Bringing them together benefits both sides. This I noticed when I worked in two separate disciplines: during my PhD at molecular haematology & oncology lab with clinician scientists and during my postdoc at BRIC, Copenhagen with scientists experts in epigenetic mechanisms.<\/span><\/p>\n

Our first seminar in December 2020 had over 500 attendees. We expected interest to decline after the pandemic, but attendance has\u00a0remained\u00a0strong. We are now entering our sixth year, with more than 10,000-15,000 people having attended\u00a0in total.<\/span>\u00a0<\/span><\/p>\n

Recently, when I went to a conference in the US, people recognised me and said their entire labs attend the seminars monthly, so it has become a valuable educational platform for those in the field.<\/span><\/p>\n

Outside of work, what are your passions and hobbies?<\/span><\/span><\/h2>\n

I\u2019m\u00a0a mother of two\u00a0young\u00a0boys, so when\u00a0I\u2019m\u00a0not working,\u00a0I\u2019m\u00a0busy with them!\u00a0<\/span>\u00a0<\/span><\/p>\n

My main hobbies are painting, baking, and I love cooking, following recipes, and trying new dishes. It gives me the same feeling as doing experiments and following protocols. I also enjoy travelling whenever possible, although that\u2019s less frequent these days. I have a keen interest in watching crime series, which motivates me to solve mysteries in my own research and think from the cancer cells’ mind to hide and escape treatment.<\/p>\n","protected":false},"excerpt":{"rendered":"

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